Eosinophils residing in the airways of mice respond to influenza A virus (IAV) infection through alterations in surface expression of various markers necessary for migration and cellular immunity responses, according to research published in the Journal of Leukocyte Biology by researchers from Le Bonheur Children’s Hospital and the University of Tennessee Health Science Center.
Le Bonheur and the University of Tennessee Health Science Center neonatologists, led by Jennifer M. Davidson, DO, conducted a randomized trial for the treatment of hemodynamically significant PDAs (hsPDAs) in very low birth weight (VLBW) infants. Echocardiogram criteria before and after treatment showed that IV indomethacin was more effective.
Jeffrey A. Towbin, MD, of Le Bonheur Children’s Hospital and the University of Tennessee Health Science Center was awarded a Research Project Grant (R01) from the National Institutes of Health (NIH) with co-principal investigators Enkhsaikha Purevjav, MD, PhD and Lu Lu, MD, MS. He is lead principal investigator on the project “Discovery of Modifier Genes in Cardiomyopathy.”
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